Prediction of Progression-Free Survival in Patients With Advanced, Well-Differentiated, Neuroendocrine Tumors Being Treated With a Somatostatin Analog: The GETNE-TRASGU Study


Por: Carmona-Bayonas A, Jiménez-Fonseca P, Lamarca Á, Barriuso J, Castaño Á, Benavent M, Alonso V, Riesco-Martínez MDC, Alonso-Gordoa T, Custodio A, Sánchez Cánovas M, Hernando Cubero J, López C, Lacasta A, Fernández Montes A, Marazuela M, Crespo G, Escudero P, Diaz JÁ, Feliciangeli E, Gallego J, Llanos M, Segura Á, Vilardell F, Percovich JC, Grande E, Capdevila J, Valle JW and García-Carbonero R

Publicada: 1 oct 2019
Resumen:
PURPOSE Somatostatin analogs (SSAs) are recommended for the first-line treatment of most patients with well-differentiated, gastroenteropancreatic (GEP) neuroendocrine tumors; however, benefit from treatment is heterogeneous. The aim of the current study was to develop and validate a progression-free survival (PFS) prediction model in SSA-treated patients. PATIENTS AND METHODS We extracted data from the Spanish Group of Neuroendocrine and Endocrine Tumors Registry (R-GETNE). Patient eligibility criteria included GEP primary, Ki-67 of 20% or less, and first-line SSA monotherapy for advanced disease. An accelerated failure time model was developed to predict PFS, which was represented as a nomogram and an online calculator. The nomogram was externally validated in an independent series of consecutive eligible patients (The Christie NHS Foundation Trust, Manchester, United Kingdom). RESULTS We recruited 535 patients (R-GETNE, n = 438; Manchester, n = 97). Median PFS and overall survival in the derivation cohort were 28.7 (95% CI, 23.8 to 31.1) and 85.9 months (95% CI, 71.5 to 96.7 months), respectively. Nine covariates significantly associated with PFS were primary tumor location, Ki-67 percentage, neutrophil-to-lymphocyte ratio, alkaline phosphatase, extent of liver involvement, presence of bone and peritoneal metastases, documented progression status, and the presence of symptoms when initiating SSA. The GETNE-TRASGU (Treated With Analog of Somatostatin in Gastroenteropancreatic and Unknown Primary NETs) model demonstrated suitable calibration, as well as fair discrimination ability with a C-index value of 0.714 (95% CI, 0.680 to 0.747) and 0.732 (95% CI, 0.658 to 0.806) in the derivation and validation series, respectively. CONCLUSION The GETNE-TRASGU evidence-based prognostic tool stratifies patients with GEP neuroendocrine tumors receiving SSA treatment according to their estimated PFS. This nomogram may be useful when stratifying patients with neuroendocrine tumors in future trials. Furthermore, it could be a valuable tool for making treatment decisions in daily clinical practice. (C) 2019 by American Society of Clinical Oncology.

Filiaciones:
Carmona-Bayonas A:
 Hospital Universitario Morales Meseguer, Universidad de Murcia, Instituto Murciano de Investigación Biosanitaria, Murcia, Spain

Jiménez-Fonseca P:
 Hospital Universitario Central de Asturias, Oviedo, Spain

Lamarca Á:
 The Christie NHS Foundation Trust, Manchester, United Kingdom

Barriuso J:
 University of Manchester, Manchester, United Kingdom

 The Christie NHS Foundation Trust, Manchester, United Kingdom

Castaño Á:
 Hospital Universitario de Fuenlabrada, Madrid, Spain

Benavent M:
 Hospital Universitario Virgen del Rocío, Instituto de Biomedicina de Sevilla, Sevilla, Spain

Alonso V:
 Hospital Universitario Miguel Servet, Zaragoza, Spain

Riesco-Martínez MDC:
 Hospital Universitario Doce de Octubre, Madrid, Spain

Alonso-Gordoa T:
 Hospital Universitario Ramón Y Cajal, Madrid, Spain

Custodio A:
 Hospital Universitario La Paz, Centro de Investigación Biomédica en Red Cáncer, CB16/12/00398, Madrid, Spain

Sánchez Cánovas M:
 Hospital Universitario Morales Meseguer, Universidad de Murcia, Instituto Murciano de Investigación Biosanitaria, Murcia, Spain

Hernando Cubero J:
 Hospital Universitario Vall d'Hebron, Vall Hebron Institute of Oncology, Autonomous University of Barcelona, Barcelona, Spain

López C:
 Hospital Universitario Marqués de Valdecilla, Santander, Spain

Lacasta A:
 Hospital Universitario Donostia, San Sebastián, Spain

Fernández Montes A:
 Complexo Hospitalario Universitario de Ourense, Orense, Spain

Marazuela M:
 Hospital Universitario de La Princesa, Madrid, Spain

Crespo G:
 Complejo Asistencial Universitario de Burgos, Burgos, Spain

Escudero P:
 Hospital Clinico Universitario Lozano Blesa, Zaragoza, Spain

Diaz JÁ:
 Hospital Universitario Clínico San Carlos, Madrid, Spain

Feliciangeli E:
 Hospital Universitario Santa Lucia, Cartagena, Spain

:
 Hospital General Universitario de Elche, Elche, Spain

Llanos M:
 Hospital Universitario de Canarias, Universidad de La Laguna, Tenerife, Spain

Segura Á:
 Hospital Universitario La Fe, Valencia, Spain

Vilardell F:
 Hospital Universitari Arnau de Vilanova, Lleida, Spain

Percovich JC:
 Hospital Universitario Gregorio Marañón, Madrid, Spain

Grande E:
 MD Anderson Cancer Center Madrid, Madrid, Spain

Capdevila J:
 Hospital Universitario Vall d'Hebron, Vall Hebron Institute of Oncology, Autonomous University of Barcelona, Barcelona, Spain

Valle JW:
 The Christie NHS Foundation Trust, Manchester, United Kingdom

 University of Manchester, Manchester, United Kingdom

García-Carbonero R:
 Hospital Universitario Doce de Octubre, Universidad Complutense de Madrid, Centro Nacional de Investigaciones Oncológicas, Centro de Investigación Biomédica en Red Cáncer, Madrid, Spain
ISSN: 0732183X





JOURNAL OF CLINICAL ONCOLOGY
Editorial
AMER SOC CLINICAL ONCOLOGY, 2318 MILL ROAD, STE 800, ALEXANDRIA, VA 22314 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 37 Número: 28
Páginas: 2571-2580
WOS Id: 000489721100008
ID de PubMed: 31390276
imagen Green Published, hybrid

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