Determination of Anti-Anisakis Simplex Antibodies and Relationship with alpha beta and gamma delta Lymphocyte Subpopulations in Patients with Crohn's Disease


Por: Benet-Campos C, Cuéllar C, García-Ballesteros C, Zamora V, Gil-Borrás R, Catalán-Serra I, López-Chuliá F and Andreu-Ballester JC

Publicada: 1 abr 2017
Resumen:
The etiology of Crohn's disease (CD) is still unknown although new theories are based on defects in innate immunity. We have previously shown a decrease in gamma delta T cells in CD patients. Previous studies have shown a high prevalence of anti-A. simplex immunoglobulins in CD patients. The diminution of gamma delta T cells in the peripheral blood and intestinal mucosa of CD patients may create a state of immunosuppression that would facilitate A. simplex infection. To study the antibody responses to Anisakis antigens in Crohn's disease patients and its relationship with alpha beta and gamma delta T cell subsets. We recruited 81 CD patients and 81 healthy controls. alpha beta and gamma delta T cell subsets and anti-A. simplex antibodies were measured. Levels of anti-A. simplex IgG and IgM were significantly increased in CD patients. Almost 20% of CD patients were positive for IgG and IgM anti-A. simplex versus only 3.7 and 2.5%, respectively, in normal subjects. However, lower specific IgA levels were observed in the group of CD patients versus healthy subjects. We found an association between CD3 + CD8 + gamma delta subset and IgM anti-A. simplex levels. In ileal cases and stricturing behavior of CD, we observed the highest levels of specific antibodies with the exception of anti-A. simplex IgA. The relationship of specific antibodies with a gamma delta T cell deficiency makes these cell candidates to play a role in the immune response against Anisakis. In addition, anti-Anisakis antibodies could be considered as markers of risk of progression in CD.

Filiaciones:
:
 Hematology Department, Arnau de Vilanova Hospital, Valencia, Spain

Cuéllar C:
 Department of Parasitology, Faculty of Pharmacy, Complutense University, Madrid, Spain

:
 Hematology Department, Arnau de Vilanova Hospital, Valencia, Spain

Zamora V:
 Department of Parasitology, Faculty of Pharmacy, Complutense University, Madrid, Spain

Gil-Borrás R:
 Digestive Department, IBD Unit, Arnau de Vilanova Hospital, Valencia, Spain

Catalán-Serra I:
 Digestive Department, IBD Unit, Arnau de Vilanova Hospital, Valencia, Spain

 Internal Medicine, Gastroenterology Department, Levanger Hospital, Helse Nord-Trondelag, Levanger, Norway

 Centre of Molecular Inflammation Research (CEMIR), Norwegian Science and Technology University (NTNU), Trondheim, Norway

:
 Hematology Department, Arnau de Vilanova Hospital, Valencia, Spain

:
 Research Department, Arnau de Vilanova Hospital, c/San Clemente 12, 46015, Valencia, Spain.
ISSN: 01632116





DIGESTIVE DISEASES AND SCIENCES
Editorial
Springer Nature, VAN GODEWIJCKSTRAAT 30, 3311 GZ DORDRECHT, NETHERLANDS, Estados Unidos America
Tipo de documento: Article
Volumen: 62 Número: 4
Páginas: 934-943
WOS Id: 000396343900017
ID de PubMed: 28168577

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