Involvement of leucocyte/endothelial cell interactions in anorexia nervosa


Por: Victor, V, Rovira-Llopis, S, Saiz-Alarcon, V, Sangueesa, M, Rojo-Bofill, L, Banuls, C, de Pablo, C, Alvarez, A, Rojo, L, Rocha, M and Hernandez-Mijares, A

Publicada: 1 jul 2015
Resumen:
BackgroundAnorexia nervosa is a common psychiatric disorder in adolescence and is related to cardiovascular complications. Our aim was to study the effect of anorexia nervosa on metabolic parameters, leucocyte-endothelium interactions, adhesion molecules and proinflammatory cytokines. Materials and MethodsThis multicentre, cross-sectional, case-control study employed a population of 24 anorexic female patients and 36 controls. We evaluated anthropometric and metabolic parameters, interactions between leucocytes polymorphonuclear neutrophils (PMN) and human umbilical vein endothelial cells (HUVEC), proinflammatory cytokines such as tumour necrosis factor alpha (TNF-) and interleukin-6 (IL-6) and soluble cellular adhesion molecules (CAMs) including E-selectin, vascular cell adhesion molecule-1 (VCAM-1) and intercellular adhesion molecule-1 (ICAM-1). ResultsAnorexia nervosa was related to a decrease in weight, body mass index, waist circumference, systolic blood pressure, glucose, insulin and HOMA-IR, and an increase in HDL cholesterol. These effects disappeared after adjusting for BMI. Anorexia nervosa induced a decrease in PMN rolling velocity and an increase in PMN rolling flux and PMN adhesion. Increases in IL-6 and TNF- and adhesion molecule VCAM-1 were also observed. ConclusionsThis study supports the hypothesis of an association between anorexia nervosa, inflammation and the induction of leucocyte-endothelium interactions. These findings may explain, in part at least, the increased risk of vascular disease among patients with anorexia nervosa.

Filiaciones:
:
 Fdn Promot Healthcare & Biomed Res Valencian Comm, Valencia, Spain

 Univ Hosp Doctor Peset, Serv Endocrinol, Valencia, Spain

 Univ Valencia, Inst Hlth Res INCLIVA, Valencia, Spain

 Univ Valencia, Dept Physiol, Valencia, Spain

:
 Fdn Promot Healthcare & Biomed Res Valencian Comm, Valencia, Spain

 Univ Hosp Doctor Peset, Serv Endocrinol, Valencia, Spain

 Univ Valencia, Inst Hlth Res INCLIVA, Valencia, Spain

Saiz-Alarcon, V:
 Univ Valencia, Psychiat Serv, Univ Hosp La Fe, Dept Med, Valencia, Spain

Sangueesa, M:
 Univ Valencia, Psychiat Serv, Univ Hosp La Fe, Dept Med, Valencia, Spain

Rojo-Bofill, L:
 Univ Valencia, Psychiat Serv, Univ Hosp La Fe, Dept Med, Valencia, Spain

:
 Fdn Promot Healthcare & Biomed Res Valencian Comm, Valencia, Spain

 Univ Hosp Doctor Peset, Serv Endocrinol, Valencia, Spain

 Univ Valencia, Inst Hlth Res INCLIVA, Valencia, Spain

:
 Univ Valencia, Dept Pharmacol, Valencia, Spain

 Univ Valencia, CIBER Res Grp CB06 04 0071, CIBER Hepat & Digest Dis, Valencia, Spain

:
 Univ Valencia, Dept Pharmacol, Valencia, Spain

 Univ Valencia, CIBER Res Grp CB06 04 0071, CIBER Hepat & Digest Dis, Valencia, Spain

 Univ Valencia, Fdn Gen, Valencia, Spain

Rojo, L:
 Univ Valencia, Psychiat Serv, Univ Hosp La Fe, Dept Med, Valencia, Spain

 Univ Valencia, Res Grp CIBER CIBER Act Epidemiol & Publ Hlth CB, Valencia, Spain

 Univ Valencia, Dept Med, Valencia, Spain

:
 Fdn Promot Healthcare & Biomed Res Valencian Comm, Valencia, Spain

 Univ Hosp Doctor Peset, Serv Endocrinol, Valencia, Spain

 Univ Valencia, Inst Hlth Res INCLIVA, Valencia, Spain

:
 Fdn Promot Healthcare & Biomed Res Valencian Comm, Valencia, Spain

 Univ Hosp Doctor Peset, Serv Endocrinol, Valencia, Spain

 Univ Valencia, Inst Hlth Res INCLIVA, Valencia, Spain

 Univ Valencia, Dept Med, Valencia, Spain
ISSN: 00142972





EUROPEAN JOURNAL OF CLINICAL INVESTIGATION
Editorial
WILEY-BLACKWELL, 111 RIVER ST, HOBOKEN 07030-5774, NJ USA, Reino Unido
Tipo de documento: Article
Volumen: 45 Número: 7
Páginas: 670-678
WOS Id: 000357031700002
ID de PubMed: 25944525

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