The use of beta-blockers is associated with a lower risk of developing hepatocellular carcinoma in patients with cirrhosis


Por: Herrera I, Pascual S, Zapater P, Carnicer F, Bellot P and Palazon J

Publicada: 1 oct 2016
Resumen:
IntroductionPatients with cirrhosis by hepatitis C virus infection treated with -blockers (BB) have been shown to have a reduced incidence of hepatocellular carcinoma (HCC). Also, an association between propranolol therapy and lower incidence of other tumors has been described.AimTo analyze the incidence of HCC according to BB treatment in cirrhosis of any cause.Patients and methodsCirrhotic patients included in the program for early detection of HCC were followed. Patients' data were prospectively registered, including transplantation and death. Patients were classified as chronically taken or not BB and the proportions of patients who remained free of tumor from the diagnosis of cirrhosis until the end of follow-up were compared using Kaplan-Meier analysis and the Breslow test.ResultsA total of 173 patients (73 treated and 100 untreated BB) were followed. The median duration of follow-up was 11 years. There were no differences between both groups in the overall survival, number of deaths, or liver transplant.Overall, 28 patients developed HCC during the follow-up, 20 patients who were untreated and eight patients treated with BB. The cumulative proportion of cases of HCC between untreated and treated with BB from the diagnosis of cirrhosis was statistically significant (6 vs. 3%, at 5 years; 19 vs. 6% at 10 years; 24 vs. 16% at 15 years; P=0.048). Multivariate analyses showed BB intake as the only significant variable associated with the development of HCC.ConclusionCirrhotic patients treated with BB have a lower cumulative probability of developing HCC during the 10 years after the diagnosis of cirrhosis.

Filiaciones:
Herrera I:
 Hosp Gen Univ Alicante, CIBEREHD, Alicante, Spain

 Hosp Gen Univ Alicante, Dept Gastroenterol, Liver Unit, C Pintor Baeza S-N, Alicante 03010, Spain

Pascual S:
 Hosp Gen Univ Alicante, CIBEREHD, Alicante, Spain

 Hosp Gen Univ Alicante, Dept Gastroenterol, Liver Unit, C Pintor Baeza S-N, Alicante 03010, Spain

Zapater P:
 Hosp Gen Univ Alicante, CIBEREHD, Alicante, Spain

 Hosp Gen Univ Alicante, Clin Pharmacol Unit, Alicante, Spain

 Univ Miguel Hernandez, Alicante, Spain

Carnicer F:
 Hosp Gen Univ Alicante, CIBEREHD, Alicante, Spain

 Hosp Gen Univ Alicante, Dept Gastroenterol, Liver Unit, C Pintor Baeza S-N, Alicante 03010, Spain

Bellot P:
 Hosp Gen Univ Alicante, CIBEREHD, Alicante, Spain

 Hosp Gen Univ Alicante, Dept Gastroenterol, Liver Unit, C Pintor Baeza S-N, Alicante 03010, Spain

Palazon J:
 Hosp Gen Univ Alicante, CIBEREHD, Alicante, Spain

 Hosp Gen Univ Alicante, Dept Gastroenterol, Liver Unit, C Pintor Baeza S-N, Alicante 03010, Spain

 Univ Miguel Hernandez, Alicante, Spain
ISSN: 0954691X





EUROPEAN JOURNAL OF GASTROENTEROLOGY & HEPATOLOGY
Editorial
LIPPINCOTT WILLIAMS & WILKINS, TWO COMMERCE SQ, 2001 MARKET ST, PHILADELPHIA, PA 19103 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 28 Número: 10
Páginas: 1194-1197
WOS Id: 000384453900013
ID de PubMed: 27294486

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