Association between phenols and thyroid hormones: The role of iodothyronine deiodinase genes


Por: Sarzo B, Abumallouh R, Marín N, Llop S, Beneito A, Lopez-Flores I, Ferrero N, Sakhi AK, Ballester F and Lopez-Espinosa MJ

Publicada: 15 oct 2022 Ahead of Print: 1 sep 2022
Resumen:
Previous literature on prenatal phenol exposure and thyroid hormone (TH) alteration is conflicting, and the possible mechanisms of action involved remain unclear. We aimed to examine the association between prenatal phenol exposure and levels of maternal and neonatal THs, as well as the possible role of iodothyronine deiodinase (DIO) gene polymorphisms in this relation.We studied 387 Spanish mother-neonate pairs with measurements of maternal phenols, total triiodothyronine (TT3) and free thyroxine (FT4), maternal and neonatal thyroid-stimulating hormone (TSH), and maternal ge-notypes for single nucleotide polymorphisms in the DIO1(rs2235544) and DIO2(rs12885300) genes. We implemented multivariate linear and weighted quantile sum (WQS) regressions to examine the association be-tween phenols and THs (including sex-stratified models for neonatal TSH) and investigated effect modification of genotypes in the maternal phenol-TH associations.In single exposure models, we found negative associations between maternal triclosan (TCS) and neonatal TSH (% change [95%CI]:-2.95 [-5.70,-0.11], per twofold phenol increase) - stronger for girls - and less clearly for maternal ethylparaben (EPB) and TSH (-2.27 [-4.55, 0.07]). In phenol mixture models, we found no association with THs. In the genetic interaction models, we found some evidence of effect modification of DIO gene poly-morphisms with stronger negative associations between methylparaben (MPB), propylparaben (PPB), butyl-paraben (BPB) and TT3 as well as bisphenol A (BPA) and FT4 for DIO1(rs2235544)-CC. Stronger inverse associations for genotypes DIO2(rs12885300)-CC and DIO2(rs12885300)-CT and positive ones for DIO2 (rs12885300)-TT were also reported for BPA and FT4.In conclusion, we found some evidence of an association between phenols and TSH during pregnancy and at birth in single exposure models, the latter being stronger for girls. Since no association was observed between maternal levels of phenols and TT3 or FT4, the possible role of the genetic background in these associations warrants further investigation.

Filiaciones:
:
 Epidemiology and Environmental Health Joint Research Unit, FISABIO-Universitat Jaume I-Universitat de València, Valencia, Spain

 Foundation for the Promotion of Health and Biomedical Research in the Valencian Region, FISABIO-Public Health, Valencia, Spain

 Department of Microbiology and Ecology, University of Valencia, Valencia, Spain

 School of Mathematics, University of Edinburgh, Edinburgh, UK

:
 Epidemiology and Environmental Health Joint Research Unit, FISABIO-Universitat Jaume I-Universitat de València, Valencia, Spain

 Foundation for the Promotion of Health and Biomedical Research in the Valencian Region, FISABIO-Public Health, Valencia, Spain

 Spanish Consortium for Research on Epidemiology and Public Health (CIBERESP), Madrid, Spain

:
 Foundation for the Promotion of Health and Biomedical Research in the Valencian Region, FISABIO-Public Health, Valencia, Spain

:
 Epidemiology and Environmental Health Joint Research Unit, FISABIO-Universitat Jaume I-Universitat de València, Valencia, Spain

 Foundation for the Promotion of Health and Biomedical Research in the Valencian Region, FISABIO-Public Health, Valencia, Spain

 Spanish Consortium for Research on Epidemiology and Public Health (CIBERESP), Madrid, Spain

:
 Epidemiology and Environmental Health Joint Research Unit, FISABIO-Universitat Jaume I-Universitat de València, Valencia, Spain

 Foundation for the Promotion of Health and Biomedical Research in the Valencian Region, FISABIO-Public Health, Valencia, Spain

Lopez-Flores I:
 Department of Genetics, Faculty of Sciences, University of Granada, Granada, Spain

Ferrero N:
 Clinical Chemistry Unit, Public Health Laboratory of Bilbao, Bizcaia, Spain

Sakhi AK:
 Norwegian Institute of Public Health, Oslo, Norway

:
 Epidemiology and Environmental Health Joint Research Unit, FISABIO-Universitat Jaume I-Universitat de València, Valencia, Spain

 Foundation for the Promotion of Health and Biomedical Research in the Valencian Region, FISABIO-Public Health, Valencia, Spain

 Spanish Consortium for Research on Epidemiology and Public Health (CIBERESP), Madrid, Spain

 Faculty of Nursing and Chiropody, University of Valencia, Valencia, Spain

:
 Epidemiology and Environmental Health Joint Research Unit, FISABIO-Universitat Jaume I-Universitat de València, Valencia, Spain

 Foundation for the Promotion of Health and Biomedical Research in the Valencian Region, FISABIO-Public Health, Valencia, Spain

 Spanish Consortium for Research on Epidemiology and Public Health (CIBERESP), Madrid, Spain

 Faculty of Nursing and Chiropody, University of Valencia, Valencia, Spain
ISSN: 02697491





ENVIRONMENTAL POLLUTION
Editorial
ELSEVIER SCI LTD, THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND, Reino Unido
Tipo de documento: Article
Volumen: 311 Número:
Páginas: 119926-119926
WOS Id: 000864075200009
ID de PubMed: 35964788

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